Zircon Lead Loss: A Kinetic Model

An age-specific kinetic model of lead metabolism in humans.

Although considerable progress has been made in recent years in reducing human exposures to lead, the potential for high intake of this contaminant still exists in millions of homes and in many occupational settings. Moreover, there is growing evidence that levels of lead intake considered inconsequential just a few years ago can result in subtle, adverse health effects, particularly in childre...

A physiologically based kinetic model for lead in children and adults.

A physiologically based model of lead kinetics in children and adults has been developed and tested. The premises on which the physiologically based model is founded are reviewed in this paper. Because 95% or more of the body burden of lead in adults is found in the bone, bone metabolism is central to the model. Bone volumes are expressed as functions of body weight. Bone formation and resorpti...

A kinetic ANNNI model

Alneact, We present a kinetic lattice system that evolves in general towards steady nonequilibrium states due to dynamical conflict between nearestand next-nearest-neighbour interactions, Under two simple particular limits, the system would reach asymptotically the canonical equi!ihium' states for the ordinary Ising model and for the axial next-nearestneighbour Ising (ANNNI) model, respectively...

Myelin loss does not lead to axonal degeneration in a long-lived model of chronic demyelination.

Current dogma suggests that chronically demyelinated axons are at risk for degeneration, with axonal loss resulting in permanent disability in myelin disease. However, the trophic role of the myelin sheath in long-term axonal survival is incompletely understood. Previous observations of the effect of dysmyelination or demyelination on axonal survival in the myelin mutants has been limited becau...

Does weight gain lead to weight loss?